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Áåñïëàòíàÿ òåõíè÷åñêàÿ áèáëèîòåêà äëÿ ëþáèòåëåé è ïðîôåññèîíàëîâ Áåñïëàòíàÿ òåõíè÷åñêàÿ áèáëèîòåêà


Àíãëèéñêèé ÿçûê äëÿ ìåäèêîâ. Air flow (ñàìîå âàæíîå)

Êîíñïåêòû ëåêöèé, øïàðãàëêè

Ñïðàâî÷íèê / Êîíñïåêòû ëåêöèé, øïàðãàëêè

Êîììåíòàðèè ê ñòàòüå Êîììåíòàðèè ê ñòàòüå

Îãëàâëåíèå (ðàçâåðíóòü)

31. Air flow

Air moves from areas of higher pressure to areas of lower pres sure just as fluids do. A pressure gradient needs to be established to move air.

Alveolar pressure becomes less than atmospheric pressure when the muscles of inspiration enlarge the chest cavity, thus lowering the intrathoracic pressure. In-trapleural pressure decreases, caus ing expansion of the alveoli and reduction of intra-alveolar pressure. The pressure gradient between the atmosphere and the alveoli drives air into the airways. The opposite occurs with expiration.

Air travels in the conducting airways via bulk flow (mL/min). Bulk flow may be turbulent or laminar, depending on its velocity. Velocity represents the speed of movement of a single particle in the bulk flow. At high velocities, the flow may be turbulent. At lower velocities transitional flow is likely to occur. At still lower velocities, flow may be laminar (streamlined). Reynold's number predicts the air flow. The higher the number, the more likely the air will be turbulent. The velocity of particle movement slows as air moves deeper into the lungs because of the enormous increase in cross-sectional area due to branching. Diffusion is the primary mechanism by which gas moves between terminal bronchioles and alveoli (the respiratory zone).

Airway resistance: The pressure difference necessary to produce gas flow is directly related to the resistance caused by friction at the airway walls. Medium-sized airways (> 2 mm diameter) are the major site of airway resistance. Small airways have a high individual resis tance. However, their total resistance is much less because resistances in parallel add as reciprocals.

Factors affecting airway resistance: Bronchocon-striction (increased resistance) can be caused by parasympathetic stimulation, histamine (immediate hyper-sensitivity reaction), slow-reacting substance of anaphyla-xis (SRS-A = leukotrienes C4, D4, E4; mediator of asthma), and irritants. Bronchodilation (decreased resistance) can be caused by sympathetic stimulation (via beta-2 receptors). Lung volume also affects airway resistance. High lung vol umes lower airway resistance because the surrounding lung parenchyma pulls airways open by radial traction. Low lung volumes lead to increased airway resistance because there is less traction on the airways. At very low lung vol umes, bronchioles may collapse. The viscosity or density of inspired gases can affect airway resistance. The density of gas increases with deep sea div ing, leading to increased resistance and work of breathing. Low-density gases like helium can lower airway resistance During a forced expiration, the airways are compressed by increased intrathoracic pressure. Regardless of how forceful the expiratory effort is, the flow rate plateaus and cannot be exceeded. Therefore, the air flow is effort-independent; the collapse of the airways is called dynamic compression. Whereas this phenomenon is seen only upon forced expira tion in normal subjects, this limited flow can be seen dur ing normal expiration in patients with lung diseases where there is increased resistance (e. g., asthma) or increased compliance (e. g., emphysema).

New words

intrapleural - âíóòðèïëåâðàëüíûé

intra-alveolar - âíóòðèàëüâåîëÿðíûé

collapse - êîëëàïñ

viscosity - âÿçêîñòü

density - ïëîòíîñòü

Àâòîð: Åëåíà Áåëèêîâà

<< Íàçàä: Ventilation

>> Âïåðåä: Mechanics of breathing

Ðåêîìåíäóåì èíòåðåñíûå ñòàòüè ðàçäåëà Êîíñïåêòû ëåêöèé, øïàðãàëêè:

Êðàòêîå ñîäåðæàíèå ïðîèçâåäåíèé ðóññêîé ëèòåðàòóðû XIX âåêà

Ñïåöèàëüíàÿ ïñèõîëîãè. Øïàðãàëêà

Ýêîíîìèêà íåäâèæèìîñòè. Øïàðãàëêà

Ñìîòðèòå äðóãèå ñòàòüè ðàçäåëà Êîíñïåêòû ëåêöèé, øïàðãàëêè.

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Ðàñêðûòà ñâÿçü ìåæäó ãîëîäîì è íàñòðîåíèåì 03.10.2018

Èññëåäîâàòåëè èç Ãóýëôñêîãî Óíèâåðñèòåòà ïîêàçàëè, ÷òî âíåçàïíîå ïàäåíèå ãëþêîçû, êîòîðîå ìû èñïûòûâàåì, êîãäà ìû ãîëîäíû, ìîæåò âëèÿòü íà íàøå íàñòðîåíèå.

"Ìû îáíàðóæèëè äîêàçàòåëüñòâà òîãî, ÷òî èçìåíåíèå óðîâíÿ ãëþêîçû ìîæåò îêàçûâàòü ïðîäîëæèòåëüíîå âëèÿíèå íà íàñòðîåíèå, - ñêàçàë ïðîôåññîð ôàêóëüòåòà ïñèõîëîãèè Ôðàí÷åñêî Ëåðè. - ß áûë íàñòðîåí ñêåïòè÷åñêè, êîãäà ëþäè ðàññêàçûâàëè ìíå, ÷òî îíè ñòàíîâÿòñÿ âîð÷ëèâûìè, åñëè íå åäÿò, íî òåïåðü ÿ ýòîìó âåðþ. Ãèïîãëèêåìèÿ - ñèëüíûé ôèçèîëîãè÷åñêèé è ïñèõîëîãè÷åñêèé ñòðåññ".

Ó÷åíûå èçó÷àëè âëèÿíèå âíåçàïíîãî ïàäåíèÿ ãëþêîçû íà ýìîöèîíàëüíîå ïîâåäåíèå, âûçûâàÿ ãèïîãëèêåìèþ ó êðûñ.

"Êîãäà ëþäè äóìàþò î ïëîõîì íàñòðîåíèè è ñòðåññå, îíè êàñàþòñÿ ïñèõîëîãè÷åñêèõ, à íå ìåòàáîëè÷åñêèõ ôàêòîðîâ, - ñêàçàë ìàãèñòð Òîìàñ Õîðìàí, âîçãëàâëÿâøèé èññëåäîâàíèå. - Íî ìû îáíàðóæèëè, ÷òî ïëîõîå ïèùåâîå ïîâåäåíèå ìîæåò îêàçûâàòü âëèÿíèå íà íàñòðîåíèå".

Êðûñàì ââîäèëè áëîêàòîð ìåòàáîëèçìà ãëþêîçû, çàñòàâëÿÿ èõ èñïûòûâàòü ãèïîãëèêåìèþ, à çàòåì ïîìåùàëè â îïðåäåëåííóþ êàìåðó. Ïîòîì íåñêîëüêèì êðûñàì ââîäèëè âîäó è ïîìåùàëè èõ â äðóãóþ êàìåðó. Êîãäà ïåðåä êðûñàìè âñòàâàë âûáîð, â êàêóþ êàìåðó âîéòè, îíè àêòèâíî èçáåãàëè êàìåðû, ãäå èñïûòûâàëè ãèïîãëèêåìèþ.

"Ïîâåäåíèå èçáåãàíèÿ ÿâëÿåòñÿ â äàííîì ñëó÷àå âûðàæåíèåì ñòðåññà è áåñïîêîéñòâà, - ñêàçàë Ëåðè. - Æèâîòíûå èçáåãàþò ýòîé ïàëàòû, ïîòîìó ÷òî ó íèõ áûë îòðèöàòåëüíûé îïûò, è îíè áîëüøå íå õîòÿò åãî ïåðåæèâàòü".

Èññëåäîâàòåëè ïðîâîäèëè àíàëèç êðîâè ó êðûñ ïîñëå âîçíèêíîâåíèÿ ãèïîãëèêåìèè è îáíàðóæèëè ïîâûøåííûé êîðòèêîñòåðîí, ÷òî ñâèäåòåëüñòâóåò î ôèçèîëîãè÷åñêîì ñòðåññå. Êðîìå òîãî, ïîëó÷àâøèå áëîêàòîð ìåòàáîëèçìà ãëþêîçû êðûñû ñòàíîâèëèñü âÿëûìè è áåçæèçíåííûìè.

"Âû ìîæåòå óòâåðæäàòü, ÷òî îíè íóæäàþòñÿ â ãëþêîçå, ÷òîáû çàñòàâèòü ìûøöû ðàáîòàòü, - ñêàçàë Ëåðè. - Íî êîãäà ìû äàâàëè èì îáùåïðèíÿòûå àíòèäåïðåññàíòû, òî âÿëîå ïîâåäåíèå èñ÷åçàëî. Æèâîòíûå ïåðåäâèãàëèñü â îáû÷íîì òåìïå. Ýòî èíòåðåñíî: èõ ìûøöû íå ïîëó÷àëè ãëþêîçó, íî ïîâåäåíèå ìåíÿëîñü".

Ýòîò âûâîä ïîäòâåðæäàåò èäåþ î òîì, ÷òî æèâîòíûå èñïûòûâàëè ñòðåññ è ïîäàâëåííîå íàñòðîåíèå â ñîñòîÿíèè ãèïîãëèêåìèè.

Äðóãèå èíòåðåñíûå íîâîñòè:

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